On June 1, 2018, the University of Geneva will open a new Doctoral School in Life Sciences shared between the Faculties of Medicine and Science, which will jointly award a Doctorate of Science. This innovative doctoral training meets international criteria of excellence, reflecting the recognized scientific excellence of the University of Geneva.
6 July 2018, afternoon
Room A150, Sciences II
Deadline for registration: June 10
Brigitte Galliot, vice-doyenne de la Faculté des sciences, a été nommée vice-rectrice de l’UNIGE. Elle prendra ses fonctions le 1er août 2018. En charge des ressources humaines et de la recherche, elle mettra sa vaste expérience au service de l’institution qu’elle a rejointe en 1993. La promotion de la recherche, la formation des chercheuses et chercheurs au management et la place des femmes dans l’univers académique font partie de ses priorités.
Plants, just like animals, contain growth promoting steroid hormones. The active hormone is sensed at the plasma membrane by a plant-unique receptor kinase. For activation, the plant steroid receptor relies on a shape-complementary helper protein. Researchers from Michael Hothorn‘s team have now uncovered that these helper proteins are targeted by other receptor kinases termed BIR proteins. The steroid receptor and BIRs compete for binding to the helper protein, thereby shaping plant growth in different cells and tissues. The study was published on May 07, 2018 in Nature Plants.
Empowering citizens, social innovation, scientific literacy
3 June: Public Citizen Science Festival
Discover local Citizen Science projects with hands on activities, and meet activists in this field from all over the world. An excursion with a Bioblitz is organized by the Bioscope.
4 – 5 June: Conference
Keynote speaker: Bruno Strasser: Citizen Science? Rethinking Science and Public Participation
The team of David Shore has shown how telomeres control the initiation of DNA replication genome-wide. The study, published on April 24, 2018 in Cell Reports, reveals that a telomere-binding protein called Rif1 is sequestered at telomeres, limiting its repressive effect on origins of DNA replication to regions near the ends of chromosomes. This may allow chromosomes ends to act as nurseries for genome evolution.